Biography
Biography: Peter Tunbridge
Abstract
Two years ago, I was asked by the Breast Cancer Society of New Zealand to propose a new model of cancer because the current model was deemed to be inadequate. This new model is based on the cancer arising in visceral fat stem cells that have been transported to the affected organ by the immune system to act as rapid glucose metabolizers. It examines the role of sex hormones and thyroid hormones in mediating this metabolic response to transient glucose overload in the lumen of the endothelial cells. The model can explain why cancer is not destroyed by the immune system and finally gives an understanding of the process of metastasis, which are not part of the original tumor but new genetically similar primaries. The model was received with great enthusiasm (standing ovation).